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NCT04602325 · ClinicalTrials.gov registry record

Systemic Biomarkers of Brain Injury From Hyperammonemia

A clinical trial of Hypoxic Ischemic Encephalopathy and Urea Cycle Disorder, sponsored by Children's National Research Institute.

Recruiting
Registry status
24
Enrollment target
1
Study location

NCT04602325 is a study of Hypoxic Ischemic Encephalopathy and Urea Cycle Disorder that is actively recruiting participants, run by Children's National Research Institute. The registered enrollment target is 24 participants, below the 81-participant average among 13 other Hypoxic Ischemic Encephalopathy trials with a reported enrollment target (70% lower). The trial reports 1 study location across 1 state.

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The verdict

NCT04602325, a study of Hypoxic Ischemic Encephalopathy and Urea Cycle Disorder, is actively recruiting participants, sponsored by Children's National Research Institute.

RECRUITING
Registry status
24 participants
Enrollment target
1
Study location

Study Summary

Ammonia is a waste product of protein and amino acid catabolism and is also a potent neurotoxin. High blood ammonia levels on the brain can manifest as cytotoxic brain edema and vascular compromise leading to intellectual and developmental disabilities. The following aims are proposed: Aim 1 of this study will be to determine the chronology of biomarkers of brain injury in response to a hyperammonemic (HA) brain insult in patients with an inherited hyperammonemic disorder. Aim 2 will be to determine if S100B, NSE, and UCHL1 are altered in patients with two other inborn errors of metabolism, Maple Syrup Urine Disease (MSUD) and Glutaric Acidemia (GA1).

Study Locations (1)

District of Columbia

  • Children's National Research Institute - Washington D.C.

Trial Details

FieldValue
Enrollment Target 24 participants
Start Date 2020-07-09
Est. Completion 2027-05

What the Registry Record Tells You About NCT04602325

The ClinicalTrials.gov registry entry for NCT04602325 describes a study currently listed as recruiting, categorized as an unspecified phase. The registered enrollment target is 24 participants, a figure that helps gauge the scale of data the investigators plan to collect, below the 81-participant average among 13 other Hypoxic Ischemic Encephalopathy trials with a reported enrollment target (70% lower). The listed sponsor is Children's National Research Institute, which has 92 total studies on file at ClinicalTrials.gov.

The record links to 6 conditions, with Hypoxic Ischemic Encephalopathy appearing as the primary indexed condition, and to 0 interventions.

NCT04602325 reports 1 study location spanning 1 distinct geographic area - top geographies include District of Columbia.

Frequently Asked Questions

What is clinical trial NCT04602325 about?

NCT04602325 is a clinical study titled "Systemic Biomarkers of Brain Injury From Hyperammonemia". Ammonia is a waste product of protein and amino acid catabolism and is also a potent neurotoxin. High blood ammonia levels on the brain can manifest as cytotoxic brain edema and vascular compromise leading to intellectual and developmental disabilities. The following aims are proposed: Aim 1 of thi...

What is the current status of trial NCT04602325?

This trial is currently recruiting. The enrollment target is 24 participants. The study started on 2020-07-09. Estimated completion is 2027-05.

What conditions does trial NCT04602325 study?

This clinical trial studies the following conditions: Hypoxic Ischemic Encephalopathy, Urea Cycle Disorder, Organic Acidemia, Maple Syrup Urine Disease, Glutaric Acidemia I.

Who is sponsoring clinical trial NCT04602325?

This trial is sponsored by Children's National Research Institute, which has 92 total clinical trials registered on ClinicalTrials.gov.

Where is trial NCT04602325 being conducted?

This trial has 1 study location across District of Columbia. Contact the study sites directly through ClinicalTrials.gov for enrollment availability.

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Data sourced from official public datasets. See our methodology for details. Retrieved and formatted by PlainTrial Editorial

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