Medical Information Only. Always consult your healthcare provider before enrolling in any clinical trial.

NCT02972554 · ClinicalTrials.gov registry record · Phase 4

Does Propranolol Attenuate Inflammatory Responses to a Psychological Stressor?

A Phase 4 study, sponsored by University of North Carolina, Chapel Hill.

Completed
Registry status
Phase 4
Development phase
92
Enrollment target

NCT02972554 is a Phase 4 study that has completed, run by University of North Carolina, Chapel Hill. The registered enrollment target is 92 participants.

View on ClinicalTrials.gov ↗

View your shortlist →

The verdict

NCT02972554, a Phase 4 study, has completed, sponsored by University of North Carolina, Chapel Hill.

COMPLETED
Registry status
Phase 4
Development phase
92 participants
Enrollment target

Study Summary

This randomized, double-blind, placebo-controlled study of propranolol will shed important light on how sympathetic nervous system (SNS) activation influences psychological and inflammatory responses to acute stress. Results from this study will inform both the basic science literature that is attempting to map the physiological mechanisms by which psychological stress may lead to poor mental and physical health, and may also ultimately have therapeutic relevance for individuals who are experiencing high levels of stress that is putting their health at risk. Utilizing a psychopharmacological approach allows for the circumvention of many of the challenges of conducting this research in human populations, and will allow for conclusions regarding causality, given that SNS activation will be experimentally manipulated, rather than relying on correlational measures of SNS activity that are difficult to assess and are not appropriate for asking if SNS activity causes changes in psychology and biology.

Interventions

  • OTHER Placebo
  • DRUG Propanolol hydrochloride

Trial Details

FieldValue
Enrollment Target 92 participants
Start Date 2016-01-26
Est. Completion 2017-10-10
Phase Phase 4

What the Registry Record Tells You About NCT02972554

The ClinicalTrials.gov registry entry for NCT02972554 describes a study currently listed as completed, categorized as Phase 4. The registered enrollment target is 92 participants, a figure that helps gauge the scale of data the investigators plan to collect. The listed sponsor is University of North Carolina, Chapel Hill, which has 1,109 total studies on file at ClinicalTrials.gov.

The record links to 0 conditions, and to 2 interventions - of which Placebo is the first listed.

NCT02972554 reports 0 study locations.

Frequently Asked Questions

What is clinical trial NCT02972554 about?

NCT02972554 is a clinical study titled "Does Propranolol Attenuate Inflammatory Responses to a Psychological Stressor?". This randomized, double-blind, placebo-controlled study of propranolol will shed important light on how sympathetic nervous system (SNS) activation influences psychological and inflammatory responses to acute stress. Results from this study will inform both the basic science literature that is attem...

What is the current status of trial NCT02972554?

This trial is currently completed. It is a Phase 4 study. The enrollment target is 92 participants. The study started on 2016-01-26. Estimated completion is 2017-10-10.

What interventions are being tested in trial NCT02972554?

The interventions under investigation include: Placebo (OTHER), Propanolol hydrochloride (DRUG).

Who is sponsoring clinical trial NCT02972554?

This trial is sponsored by University of North Carolina, Chapel Hill, which has 1,109 total clinical trials registered on ClinicalTrials.gov.

Data sourced from official public datasets. See our methodology for details. Retrieved and formatted by PlainTrial

Every figure on PlainTrial is rendered directly from the ClinicalTrials.gov registry, no number is typed in by an editor. This page mirrors this trial's own ClinicalTrials.gov registry record, live from the dataset. See our editorial standards & corrections policy, the methodology behind these numbers, or report a data error.