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NCT01348204 · ClinicalTrials.gov registry record · Phase 2

Nasal Potential Studies Utilizing Cystic Fibrosis Transmembrane Regulator (CFTR) Modulators

A Phase 2 study of Cystic Fibrosis, sponsored by University of Alabama at Birmingham.

Completed
Registry status
Phase 2
Development phase
32
Enrollment target
1
Study location

NCT01348204: Completed Phase 2 study of Cystic Fibrosis, sponsored by University of Alabama at Birmingham.

NCT01348204 is a Phase 2 study of Cystic Fibrosis that has completed, run by University of Alabama at Birmingham. The registered enrollment target is 32 participants, below the 179-participant average among 169 other Cystic Fibrosis trials with a reported enrollment target (82% lower). The trial reports 1 study location across 1 state. According to ClinicalTrials.gov, the official US trial registry.

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The verdict

NCT01348204, a Phase 2 study of Cystic Fibrosis, has completed, sponsored by University of Alabama at Birmingham.

COMPLETED
Registry status
Phase 2
Development phase
32 participants
Enrollment target
1
Study location

Study Summary

The purpose of the study is to develop new biomarkers for studies of cystic fibrosis (CF). Defects in the gene encoding Cystic Fibrosis Transmembrane Regulator (CFTR) cause CF, an autosomal recessive disorder affecting mainly the pulmonary and digestive tract, leading to early death largely due to progressive loss of pulmonary function. In vitro experiments show that quercetin - a dietary supplement with a well-established safety profile for human use, including clinical trials in a variety of disorders encompassing cancer, heart disease, and as an anti-inflammatory agent - induces activation of CFTR. The nasal potential difference (NPD) test is a measurement of voltage across the nasal membrane and as a fundamental biomarker for CFTR activity in vivo. The NPD is a useful, well-established tool in CF research to determine both diagnoses as well as to measure the effect of new therapies. In vitro experiments show that quercetin induces activation of CFTR additive to that seen with current NPD reagents. In addition, it activates rescued mutant CFTR in vitro (∆F508 CFTR the most common cause of CF), whereas conventional agonists do not. Preliminary in vivo experiments mirrored these results and show that quercetin activates CFTR in human (n=12) NPD tests. Importantly, quercetin perfusion was well-tolerated by a validated sinus questionnaire and physician assessed nasal examination rating. These studies provide strong support for use of quercetin as potentiator of CFTR Cl- channel function by nasal administration. By adding quercetin to the sequence of perfusion solutions for NPD, the investigators may be better suited to detect ∆F508 CFTR activity of rescued mutant protein in the CF patient population.

Primary Outcome

Determine whether the NPD biomarker can be improved by including the potentiator quercetin to activate CFTR dependent ion channel activity among CF individuals with surface localized CFTR mutations

Conditions Studied

Interventions

  • OTHER quercetin

Study Locations (1)

Alabama

  • University of Alabama at Birmingham - Birmingham

Trial Details

FieldValue
Enrollment Target 32 participants
Start Date 2010-03
Est. Completion 2011-11
Phase Phase 2
University of Alabama at Birmingham

1,160 total trials

What the finished NCT01348204 record still lists

NCT01348204 is an interventional study that assigns participants to a tested intervention. The registry caps enrollment at 32 participants, a relatively small participant target, below the 179-participant average among 169 other Cystic Fibrosis trials with a reported enrollment target (82% lower).

The record links to 1 condition, with Cystic Fibrosis appearing as the primary indexed condition, and to 1 intervention - of which quercetin is the first listed.

NCT01348204 reports a single indexed study location in Alabama.

Frequently Asked Questions

What is clinical trial NCT01348204 about?

NCT01348204 is a clinical study titled "Nasal Potential Studies Utilizing Cystic Fibrosis Transmembrane Regulator (CFTR) Modulators". The purpose of the study is to develop new biomarkers for studies of cystic fibrosis (CF). Defects in the gene encoding Cystic Fibrosis Transmembrane Regulator (CFTR) cause CF, an autosomal recessive disorder affecting mainly the pulmonary and digestive tract, leading to early death largely due to p...

What is the current status of trial NCT01348204?

This trial is currently completed. It is a Phase 2 study. The enrollment target is 32 participants. The study started on 2010-03. Estimated completion is 2011-11.

What conditions does trial NCT01348204 study?

This clinical trial studies the following conditions: Cystic Fibrosis.

What interventions are being tested in trial NCT01348204?

The interventions under investigation include: quercetin (OTHER).

Who is sponsoring clinical trial NCT01348204?

This trial is sponsored by University of Alabama at Birmingham, which has 1,160 total clinical trials registered on ClinicalTrials.gov.

Where is trial NCT01348204 being conducted?

This trial has 1 study location across Alabama. Contact the study sites directly through ClinicalTrials.gov for enrollment availability.

Similar trials for Cystic Fibrosis

Matched on the same primary condition, ranked to surface studies in the same phase first, then by recruiting status, no relevance scoring or editorial curation.

Where NCT01348204's enrollment target sits among peer trials

32 111th of 169 higher than 58 of 169 other Cystic Fibrosis trials

participants (enrollment target), bucketed by value

Each bar is a band; taller bars hold more other Cystic Fibrosis trials. The dashed line + filled bar mark this entry. Hover or tap any bar for its full count and share, and where it sits relative to this entry.

Source ClinicalTrials.gov registry export · 2026-08-08

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Source: ClinicalTrials.gov NCT01348204, the US trial registry maintained by the National Library of Medicine. NCT01348204 (small enrollment · single site footprint · completed) retrieved and formatted by PlainTrial, see methodology.