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NCT00607672 · ClinicalTrials.gov registry record · Phase 4

The RAS, Fibrinolysis and Cardiopulmonary Bypass

A Phase 4 study, sponsored by Vanderbilt University.

Completed
Registry status
Phase 4
Development phase
111
Enrollment target

NCT00607672: Completed Phase 4 study, sponsored by Vanderbilt University.

NCT00607672 is a Phase 4 study that has completed, run by Vanderbilt University. The registered enrollment target is 111 participants, below the 518-participant average among 9,235 other Phase 4 trials with a reported enrollment target (79% lower). According to ClinicalTrials.gov, the official US trial registry.

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The verdict

NCT00607672, a Phase 4 study, has completed, sponsored by Vanderbilt University.

COMPLETED
Registry status
Phase 4
Development phase
111 participants
Enrollment target

Study Summary

Each year over a million patients worldwide undergo cardiac surgery requiring cardiopulmonary bypass (CPB).1 CPB is associated with significant morbidity including hemodynamic instability, the transfusion of allogenic blood products, and inflammation. Blood product transfusion increases mortality after cardiac surgery. Enhanced fibrinolysis contributes to increased blood product transfusion requirements in the perioperative period. CPB activates the kallikrein-kinin system (KKS), leading to increased bradykinin concentrations. Bradykinin, acting through its B2 receptor, stimulates the release of nitric oxide, inflammatory cytokines and tissue-type plasminogen activator (t-PA). Based on data indicating that angiotensin-converting enzyme (ACE) inhibitors reduce mortality in patients with coronary artery disease, many patients undergoing CPB are taking ACE inhibitors. While interruption of the renin-angiotensin system (RAS) reduces inflammation in response to CPB, ACE inhibitors also potentiate the effects of bradykinin and may augment B2-mediated change in fibrinolytic balance and inflammation. In contrast, angiotensin II type 1 receptor antagonism does not potentiate bradykinin and does not inhibit bradykinin metabolism. Studies in animals suggest that bradykinin receptor antagonism inhibits reperfusion-induced increases in vascular permeability and neutrophil recruitment.A randomized, placebo controlled clinical trial of a bradykinin B2 receptor antagonist demonstrated some effect on survival in patients with systemic inflammatory response syndrome and gram-negative sepsis. In addition, we and others have shown bradykinin B2 receptor antagonism reduces vascular t-PA release during ACE inhibition. The current proposal derives from data from our laboratory and others elucidating the role of the KKS in the inflammatory, hypotensive and fibrinolytic response to CPB. Specifically, we have found that CPB activates the KKS and that ACE inhibition and smoking further incre

Primary Outcome

To compare the effects of angiotensin II type I (AT1) receptor antagonism or angiotensin-converting enzyme (ACE) inhibition versus placebo on the fibrinolytic responses to cardiopulmonary bypass (CPB) as measured by t-PA antigen response

Interventions

  • DRUG Placebo
  • DRUG Ramipril
  • DRUG Candesartan

Trial Details

FieldValue
Enrollment Target 111 participants
Start Date 2006-08
Est. Completion 2011-12
Phase Phase 4
Vanderbilt University

430 total trials

What the finished NCT00607672 record still lists

NCT00607672 is an interventional study that assigns participants to a tested intervention. The registered 111 participants enrollment target is mid-sized for trials with a published cap, below the 518-participant average among 9,235 other Phase 4 trials with a reported enrollment target (79% lower).

The record links to 0 conditions, and to 3 interventions - of which Placebo is the first listed.

NCT00607672 does not publish any study locations in the registry export this page uses.

Frequently Asked Questions

What is clinical trial NCT00607672 about?

NCT00607672 is a clinical study titled "The RAS, Fibrinolysis and Cardiopulmonary Bypass". Each year over a million patients worldwide undergo cardiac surgery requiring cardiopulmonary bypass (CPB).1 CPB is associated with significant morbidity including hemodynamic instability, the transfusion of allogenic blood products, and inflammation. Blood product transfusion increases mortality af...

What is the current status of trial NCT00607672?

This trial is currently completed. It is a Phase 4 study. The enrollment target is 111 participants. The study started on 2006-08. Estimated completion is 2011-12.

What interventions are being tested in trial NCT00607672?

The interventions under investigation include: Placebo (DRUG), Ramipril (DRUG), Candesartan (DRUG).

Who is sponsoring clinical trial NCT00607672?

This trial is sponsored by Vanderbilt University, which has 430 total clinical trials registered on ClinicalTrials.gov.

How this trial's enrollment target compares

Where NCT00607672's enrollment target sits among peer trials

111 726th of 2000 higher than 1,274 of 2,000 other Phase 4 trials

participants (enrollment target), bucketed by value

Each bar is a band; taller bars hold more other Phase 4 trials. The dashed line + filled bar mark this entry. Hover or tap any bar for its full count and share, and where it sits relative to this entry.

Source ClinicalTrials.gov registry export · 2026-08-08

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Source: ClinicalTrials.gov NCT00607672, the US trial registry maintained by the National Library of Medicine. NCT00607672 (mid enrollment · none site footprint · completed) retrieved and formatted by PlainTrial, see methodology.